Glucagon Protects Against Impaired NMDA-Mediated Cerebrovasodilation and Cerebral Autoregulation during Hypotension after Brain Injury by Activating cAMP Protein Kinase A and Inhibiting Upregulation of tPA-Reference-Cited by-同舟云学术

Glucagon Protects Against Impaired NMDA-Mediated Cerebrovasodilation and Cerebral Autoregulation during Hypotension after Brain Injury by Activating cAMP Protein Kinase A and Inhibiting Upregulation of tPA

Published:2011-03 Issue:3 Volume:28 Page:451-457
ISSN:0897-7151
Container-title:Journal of Neurotrauma
language:en
Short-container-title:Journal of Neurotrauma

Author:

Armstead William M.¹²,Kiessling J. Willis¹,Cines Douglas B.³,Higazi Abd Al-Roof³⁴

Affiliation:

1. Department of Anesthesiology and Critical Care, University of Pennsylvania, Philadelphia, Pennsylvania.

2. Department of Pharmacology, University of Pennsylvania, Philadelphia, Pennsylvania.

3. Department of Pathology, University of Pennsylvania, Philadelphia, Pennsylvania.

4. Department of Clinical Biochemistry, Hadassah University Hospital, and Hebrew University-Hadassah Medical School, Jerusalem, Israel.

Publisher

Mary Ann Liebert Inc

Subject

Neurology (clinical)

Link

http://www.liebertpub.com/doi/pdf/10.1089/neu.2010.1659

Reference25 articles.

1. Age dependent NMDA contribution to impaired hypotensive cerebral hemodynamics following brain injury

2. uPA Modulates the Age-Dependent Effect of Brain Injury on Cerebral Hemodynamics through LRP and ERK MAPK

3. Plasminogen activators contribute to age-dependent impairment of NMDA cerebrovasodilation after brain injury

4. Role of impaired cAMP and calcium-sensitive K+ channel function in altered cerebral hemodynamics following brain injury

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