Redox Control of EBV Infection: Prevention by Thiol-Dependent Modulation of Functional CD21/EBV Receptor Expression-Reference-Cited by-同舟云学术

Redox Control of EBV Infection: Prevention by Thiol-Dependent Modulation of Functional CD21/EBV Receptor Expression

Published:2001-12 Issue:6 Volume:3 Page:1075-1087
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Nishinaka Yumiko¹,Nakamura Hajime¹,Okada Noriko²,Okada Hidechika²,Yodoi Junji¹

Affiliation:

1. Department of Biological Responses, Institute for Virus Research, Kyoto University, 53 Shogoin-Kawaharacho, Sakyo, Kyoto 606-8507, Japan

2. Department of Molecular Biology, Nagoya City University School of Medicine, Mizuho-cho, Nagoya 467-8601, Japan

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/152308601317203585

Reference46 articles.

1. Structure and Function of the Complement Receptors, CR1 (CD35) and CR2 (CD21)

2. Defective Regulation of Epstein–Barr Virus Infection in Patients with Acquired Immunodeficiency Syndrome (AIDS) or AIDS-Related Disorders

3. N-acetylcysteine replenishes glutathione in HIV infection

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4. Redox regulation of CD21 shedding involves signaling via PKC and indicates the formation of a juxtamembrane stalk;Journal of Cell Science;2006-07-15

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