Ischemic Postconditioning: From Receptor to End-Effector-Reference-Cited by-同舟云学术

Ischemic Postconditioning: From Receptor to End-Effector

Published:2011-03 Issue:5 Volume:14 Page:821-831
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Cohen Michael V.¹²,Downey James M.¹

Affiliation:

1. Department of Physiology, College of Medicine, University of South Alabama, Mobile, Alabama.

2. Department of Medicine, College of Medicine, University of South Alabama, Mobile, Alabama.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2010.3318

Reference77 articles.

1. Postconditioning Inhibits Mitochondrial Permeability Transition

2. The molecular composition of the mitochondrial permeability transition pore

3. Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death

4. Acidosis, oxygen, and interference with mitochondrial permeability transition pore formation in the early minutes of reperfusion are critical to postconditioning’s success

5. The pH Hypothesis of Postconditioning

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