Both Maximal Expression of Selenoproteins and Selenoprotein Deficiency Can Promote Development of Type 2 Diabetes-Like Phenotype in Mice-Reference-Cited by-同舟云学术

Both Maximal Expression of Selenoproteins and Selenoprotein Deficiency Can Promote Development of Type 2 Diabetes-Like Phenotype in Mice

Published:2011-06-15 Issue:12 Volume:14 Page:2327-2336
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Labunskyy Vyacheslav M.¹,Lee Byung Cheon¹,Handy Diane E.¹,Loscalzo Joseph¹,Hatfield Dolph L.²,Gladyshev Vadim N.¹³

Affiliation:

1. Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts.

2. Molecular Biology of Selenium Section, Laboratory of Cancer Prevention, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland.

3. Redox Biology Center and Department of Biochemistry, University of Nebraska, Lincoln, Nebraska.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2010.3526

Reference40 articles.

1. Evidence for specific selenium target tissues and new biologically important selenoproteins

2. Selenium and Diabetes: More Bad News for Supplements

3. Nutrition Intervention Trials in Linxian, China: Supplementation With Specific Vitamin/Mineral Combinations, Cancer Incidence, and Disease-Specific Mortality in the General Population

4. Mouse models for assessing the role of selenoproteins in health and development

5. Overexpression of Cellular Glutathione Peroxidase Does Not Affect Expression of Plasma Glutathione Peroxidase or Phospholipid Hydroperoxide Glutathione Peroxidase in Mice Offered Diets Adequate or Deficient in Selenium

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