Sustained Activation of Nuclear Erythroid 2-Related Factor 2/Antioxidant Response Element Signaling Promotes Reductive Stress in the Human Mutant Protein Aggregation Cardiomyopathy in Mice-Reference-Cited by-同舟云学术

Sustained Activation of Nuclear Erythroid 2-Related Factor 2/Antioxidant Response Element Signaling Promotes Reductive Stress in the Human Mutant Protein Aggregation Cardiomyopathy in Mice

Published:2011-03-15 Issue:6 Volume:14 Page:957-971
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Rajasekaran Namakkal Soorappan¹,Varadharaj Saradhadevi²,Khanderao Gayatri D.¹,Davidson Christopher J.¹,Kannan Sankaranarayanan³,Firpo Matthew A.⁴,Zweier Jay L.²,Benjamin Ivor J.¹

Affiliation:

1. Division of Cardiology, Department of Internal Medicine, University of Utah Health Sciences Center, Salt Lake City, Utah.

2. Davis Heart & Lung Research Institute, The Ohio State University, Columbus, Ohio.

3. Department of Pediatric Research, The University of Texas M. D. Anderson Cancer Center, Houston, Texas.

4. Department of General Surgery, University of Utah Health Sciences Center, Salt Lake City, Utah.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2010.3587

Reference50 articles.

1. Endogenous Methylarginines Modulate Superoxide as Well as Nitric Oxide Generation from Neuronal Nitric-oxide Synthase

2. Intrasarcoplasmic Amyloidosis Impairs Proteolytic Function of Proteasomes in Cardiomyocytes by Compromising Substrate Uptake

3. Direct Interaction between Nrf2 and p21Cip1/WAF1 Upregulates the Nrf2-Mediated Antioxidant Response

4. DJ-1, a cancer- and Parkinson's disease-associated protein, stabilizes the antioxidant transcriptional master regulator Nrf2

5. Involvement of oxidative stress and NADPH oxidase activation in the development of cardiovascular complications in a model of insulin resistance, the fructose-fed rat

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