Cardioprotection by Endoplasmic Reticulum Stress–Induced Autophagy-Reference-Cited by-同舟云学术

Cardioprotection by Endoplasmic Reticulum Stress–Induced Autophagy

Published:2011-06 Issue:11 Volume:14 Page:2191-2200
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Petrovski Goran¹,Das Somak²,Juhasz Bela³,Kertesz Attila⁴,Tosaki Arpad³,Das Dipak K.²

Affiliation:

1. Department of Biochemistry and Molecular Biology, The Apoptosis and Genomics Research Group of the Hungarian Academy of Sciences, Medical and Health Science Center, University of Debrecen, Hungary.

2. Cardiovascular Research Center, University of Connecticut School of Medicine, Farmington, Connecticut.

3. Department of Pharmacology, Faculty of Pharmacy, Medical and Health Science Center, University of Debrecen, Hungary.

4. Institute of Cardiology, Medical and Health Science Center, University of Debrecen, Hungary.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2010.3486

Reference28 articles.

1. The endoplasmic reticulum: a multifunctional signaling organelle

2. Autophagy Counterbalances Endoplasmic Reticulum Expansion during the Unfolded Protein Response

3. Myocardial ischemia and reperfusion injury

4. Potentiation of a Survival Signal in the Ischemic Heart by Resveratrol through p38 Mitogen-Activated Protein Kinase/Mitogen- and Stress-Activated Protein Kinase 1/cAMP Response Element-Binding Protein Signaling

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