Base Excision Repair and Lesion-Dependent Subpathways for Repair of Oxidative DNA Damage-Reference-Cited by-同舟云学术

Base Excision Repair and Lesion-Dependent Subpathways for Repair of Oxidative DNA Damage

Published:2011-06-15 Issue:12 Volume:14 Page:2491-2507
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Svilar David¹²,Goellner Eva M.¹²,Almeida Karen H.³,Sobol Robert W.¹²⁴

Affiliation:

1. Department of Pharmacology & Chemical Biology, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania.

2. University of Pittsburgh Cancer Institute, Hillman Cancer Center, Pittsburgh, Pennsylvania.

3. Department of Physical Sciences, Rhode Island College, Providence, Rhode Island.

4. Department of Human Genetics, University of Pittsburgh Graduate School of Public Health, Pittsburgh, Pennsylvania.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2010.3466

Reference98 articles.

1. Direct interaction between XRCC1 and UNG2 facilitates rapid repair of uracil in DNA by XRCC1 complexes

2. Mitochondrial and microsomal derived reactive oxygen species mediate apoptosis induced by transforming growth factor-?1 in immortalized rat hepatocytes

3. A unified view of base excision repair: Lesion-dependent protein complexes regulated by post-translational modification

4. Increased Specificity and Efficiency of Base Excision Repair Through Complex Formation

5. Oxidative stress and lipid peroxidation-derived DNA-lesions in inflammation driven carcinogenesis

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