Endoplasmic Reticulum Stress, Redox, and a Proinflammatory Environment in Athero-Susceptible Endothelium In Vivo at Sites of Complex Hemodynamic Shear Stress-Reference-Cited by-同舟云学术

Endoplasmic Reticulum Stress, Redox, and a Proinflammatory Environment in Athero-Susceptible Endothelium In Vivo at Sites of Complex Hemodynamic Shear Stress

Published:2011-09 Issue:5 Volume:15 Page:1427-1432
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Davies Peter F.¹²,Civelek Mete¹³

Affiliation:

1. Institute for Medicine & Engineering, University of Pennsylvania, Philadelphia Pennsylvania.

2. Department of Pathology & Laboratory Medicine, University of Pennsylvania, Philadelphia Pennsylvania.

3. Department of Medicine, University of California, Los Angeles, California.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2010.3741

Reference24 articles.

1. Chronic Endoplasmic Reticulum Stress Activates Unfolded Protein Response in Arterial Endothelium in Regions of Susceptibility to Atherosclerosis

2. Redox Control of Endoplasmic Reticulum Function

3. Coordination of ER and oxidative stress signaling: The PERK/Nrf2 signaling pathway

4. Nrf2 Is a Direct PERK Substrate and Effector of PERK-Dependent Cell Survival

5. Hemodynamic shear stress and the endothelium in cardiovascular pathophysiology

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