Oxidative Stress Stimulates Autophagic Flux During Ischemia/Reperfusion-Reference-Cited by-同舟云学术

Oxidative Stress Stimulates Autophagic Flux During Ischemia/Reperfusion

Published:2011-06 Issue:11 Volume:14 Page:2179-2190
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Hariharan Nirmala¹,Zhai Peiyong¹,Sadoshima Junichi¹

Affiliation:

1. Department of Cell Biology and Molecular Medicine, Cardiovascular Research Institute, UMDNJ, New Jersey Medical School, New Jersey.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2010.3488

Reference33 articles.

1. Upregulation of Nox4 by Hypertrophic Stimuli Promotes Apoptosis and Mitochondrial Dysfunction in Cardiac Myocytes

2. p62/SQSTM1 forms protein aggregates degraded by autophagy and has a protective effect on huntingtin-induced cell death

3. Low-pressure reperfusion alters mitochondrial permeability transition

4. Cyclophilin D is required for mitochondrial removal by autophagy in cardiac cells

5. Myocardial protection against reperfusion injury: The cGMP pathway

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