Paraoxonase 2 Deficiency Alters Mitochondrial Function and Exacerbates the Development of Atherosclerosis-Reference-Cited by-同舟云学术

Paraoxonase 2 Deficiency Alters Mitochondrial Function and Exacerbates the Development of Atherosclerosis

Published:2011-02 Issue:3 Volume:14 Page:341-351
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Devarajan Asokan¹,Bourquard Noam¹²,Hama Susan¹,Navab Mohamad¹,Grijalva Victor R.¹,Morvardi Susan³,Clarke Catherine F.³,Vergnes Laurent⁴,Reue Karen⁴,Teiber John F.⁵,Reddy Srinivasa T.¹²

Affiliation:

1. Department of Medicine, University of California, Los Angeles, California.

2. Department of Molecular and Medical Pharmacology, University of California, Los Angeles, California.

3. Department of Chemistry and Biochemistry, University of California, Los Angeles, California.

4. Department of Human Genetics, University of California, Los Angeles, California.

5. Division of Epidemiology, Department of Internal Medicine, The University of Texas Southwestern Medical Center, Dallas, Texas.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2010.3430

Reference33 articles.

1. Mitochondrial free radical generation, oxidative stress, and aging11This article is dedicated to the memory of our dear friend, colleague, and mentor Lars Ernster (1920–1998), in gratitude for all he gave to us.

2. A high diffusion coefficient for coenzyme Q10might be related to a folded structure

3. Human paraoxonases (PON1, PON2, and PON3) are lactonases with overlapping and distinct substrate specificities

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