The Role of Oxidants in Percutaneous Coronary Intervention-Induced Endothelial Dysfunction: Can We Harness Redox Signaling to Improve Clinical Outcomes?-Reference-Cited by-同舟云学术

The Role of Oxidants in Percutaneous Coronary Intervention-Induced Endothelial Dysfunction: Can We Harness Redox Signaling to Improve Clinical Outcomes?

Published:2023-05-01 Issue:13-15 Volume:38 Page:1022-1040
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Wolhuter Kathryn¹²^ORCID,Kong Stephanie M.Y.¹^ORCID,Stanley Christopher P.³^ORCID,Kovacic Jason C.¹⁴⁵^ORCID

Affiliation:

1. Victor Chang Cardiac Research Institute, Darlinghurst, New South Wales, Australia.

2. School of Biomedical Sciences, University of New South Wales, Sydney, New South Wales, Australia.

3. Heart Research Institute, University of Sydney, Sydney, Australia.

4. Zena and Michael A. Wiener Cardiovascular Institute, Icahn School of Medicine at Mount Sinai, New York, New York, USA.

5. St Vincent's Clinical School, University of New South Wales, Sydney, New South Wales, Australia.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2022.0204

Reference151 articles.

1. S-Glutathiolation by peroxynitrite activates SERCA during arterial relaxation by nitric oxide

2. TNF-α-activated eNOS signaling increases leukocyte adhesion through the S-nitrosylation pathway

3. Oxygen free radicals and systemic autoimmunity

4. The relationships between cytochromes P450 and H 2 O 2 : Production, reaction, and inhibition

5. N-Acetylcysteine as an antioxidant and disulphide breaking agent: the reasons why

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