Restoration of c-Src/Fyn Proteins Rescues Mitochondrial Dysfunction in Huntington's Disease-Reference-Cited by-同舟云学术

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Restoration of c-Src/Fyn Proteins Rescues Mitochondrial Dysfunction in Huntington's Disease

Published:2023-01-01 Issue:1-3 Volume:38 Page:95-114
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Fão Lígia¹²,Coelho Patrícia¹,Duarte Luís¹,Vilaça Rita¹³,Hayden Michael R.⁴,Mota Sandra I.¹³,Rego Ana Cristina¹²^ORCID

Affiliation:

1. Center for Neuroscience and Cell Biology (CNC), University of Coimbra, Coimbra, Portugal.

2. Faculty of Medicine, University of Coimbra, Coimbra, Portugal.

3. Institute for Interdisciplinary Research (IIIUC), University of Coimbra, Coimbra, Portugal.

4. Centre for Molecular Medicine and Therapeutics, Child and Family Research Institute, The University of British Columbia, Vancouver, Canada.

Publisher

Mary Ann Liebert Inc

Subject

Plant Science,Forestry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2022.0001

Reference45 articles.

1. Localization of PTP-1B, SHP-2, and Src Exclusively in Rat Brain Mitochondria and Functional Consequences

2. The Src non-receptor tyrosine kinase paradigm: New insights into mammalian Sertoli cell biology

3. Mutant huntingtin directly increases susceptibility of mitochondria to the calcium-induced permeability transition and cytochrome c release

4. Aggregation of Huntingtin in Neuronal Intranuclear Inclusions and Dystrophic Neurites in Brain

5. Assessing Mitochondrial Function in In Vitro and Ex Vivo Models of Huntington’s Disease

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