mTORC2 Signaling Regulates Nox4-Induced Podocyte Depletion in Diabetes-Reference-Cited by-同舟云学术

mTORC2 Signaling Regulates Nox4-Induced Podocyte Depletion in Diabetes

Published:2016-11 Issue:13 Volume:25 Page:703-719
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Eid Stéphanie¹²,Boutary Suzan¹,Braych Kawthar¹,Sabra Ramzi³,Massaad Charbel²,Hamdy Ahmed⁴,Rashid Awad⁴,Moodad Sarah¹,Block Karen⁵,Gorin Yves⁵,Abboud Hanna E.⁵,Eid Assaad A.¹

Affiliation:

1. Department of Anatomy, Cell Biology and Physiological Sciences, Faculty of Medicine and Medical Center, American University of Beirut, Beirut, Lebanon.

2. UMR-S 1124 INSERM, Paris Descartes University, Sorbonne Paris Cite University, Centre Interdisciplinaire Chimie Biology, Paris, France.

3. Department of Pharmacology and Toxicology, Faculty of Medicine and Medical Center, American University of Beirut, Beirut, Lebanon.

4. Department of Nephrology, Hamad Medical Corporation, Doha, Qatar.

5. Department of Medicine, South Texas Veterans Healthcare System and the University of Texas Health Science Center, San Antonio, Texas.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2015.6562

Reference57 articles.

1. Mesangial cell biology

2. Mouse models of diabetic nephropathy

3. The NOX Family of ROS-Generating NADPH Oxidases: Physiology and Pathophysiology

4. Subcellular localization of Nox4 and regulation in diabetes

5. AKT2 is essential to maintain podocyte viability and function during chronic kidney disease

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