Control of Oxidative Stress and Inflammation in Sickle Cell Disease with the Nrf2 Activator Dimethyl Fumarate-Reference-Cited by-同舟云学术

Control of Oxidative Stress and Inflammation in Sickle Cell Disease with the Nrf2 Activator Dimethyl Fumarate

Published:2017-05-10 Issue:14 Volume:26 Page:748-762
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Belcher John D.¹,Chen Chunsheng¹,Nguyen Julia¹,Zhang Ping¹,Abdulla Fuad¹,Nguyen Phong¹,Killeen Trevor¹,Xu Pauline¹,O'Sullivan Gerry²,Nath Karl A.³,Vercellotti Gregory M.¹

Affiliation:

1. Division of Hematology, Oncology and Transplantation, Department of Medicine, Vascular Biology Center, University of Minnesota, Minneapolis, Minnesota.

2. Veterinary Population Medicine, University of Minnesota, St. Paul, Minnesota.

3. Division of Nephrology and Hypertension, Department of Medicine, Mayo Clinic/Foundation, Rochester, Minnesota.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2015.6571

Reference65 articles.

1. Effects of dimethyl fumarate on neuroprotection and immunomodulation

2. Protective Role of Hemopexin on Heme-Dependent Lung Oxidative Stress

3. Heme oxygenase-1 gene promoter polymorphism is associated with reduced incidence of acute chest syndrome among children with sickle cell disease

4. Heme triggers TLR4 signaling leading to endothelial cell activation and vaso-occlusion in murine sickle cell disease

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