NADPH Oxidase NOX4 Is a Critical Mediator of BRAFV600E-Induced Downregulation of the Sodium/Iodide Symporter in Papillary Thyroid Carcinomas-Reference-Cited by-同舟云学术

NADPH Oxidase NOX4 Is a Critical Mediator of BRAFV600E-Induced Downregulation of the Sodium/Iodide Symporter in Papillary Thyroid Carcinomas

Published:2017-05-20 Issue:15 Volume:26 Page:864-877
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Azouzi Naïma¹²³⁴,Cailloux Jérémy¹²³,Cazarin Juliana M.¹²³⁵,Knauf Jeffrey A.⁶,Cracchiolo Jennifer⁶,Al Ghuzlan Abir¹²³,Hartl Dana²,Polak Michel⁷⁸⁹¹⁰,Carré Aurore⁷⁸,El Mzibri Mohammed⁴,Filali-Maltouf Abdelkarim¹¹,Al Bouzidi Abderrahmane¹²,Schlumberger Martin¹²³,Fagin James A.⁶,Ameziane-El-Hassani Rabii¹²⁴,Dupuy Corinne¹²³

Affiliation:

1. UMR 8200 CNRS, Villejuif, France.

2. Institut Gustave Roussy, Villejuif, France.

3. Université Paris-Saclay, Orsay, France.

4. Unité de Biologie et Recherche Médicale, Centre National de l'Energie, des Sciences et des Techniques Nucléaires, Rabat, Morocco.

5. Laboratório de Fisiologia Endócrina Doris Rosenthal, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, Rio de Janeiro, Brazil.

6. Department of Medicine and Human Oncology and Pathogenesis Program, Memorial Sloan-Kettering Cancer Center, New York, New York.

7. INSERM U1016, Paris, France.

8. Imagine Institute, Paris, France.

9. Pediatric Endocrinology, Gynaecology and Diabetology Unit, Hôpital Universitaire Necker-Enfants Malades, AP-HP, Paris, France.

10. Université Paris Descartes–Sorbonne Paris Cité, Paris, France.

11. Laboratoire de Microbiologie et Biologie Moléculaire, Faculté des Sciences, Université Mohammed V, Rabat, Morocco.

12. Equipe de recherche en pathologie tumorale, Faculté de Médecine et de Pharmacie, Université Mohammed V, Rabat, Morocco.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2015.6616

Reference53 articles.

1. Direct Interaction of the Novel Nox Proteins with p22phox Is Required for the Formation of a Functionally Active NADPH Oxidase

2. NADPH oxidase DUOX1 promotes long-term persistence of oxidative stress after an exposure to irradiation

3. Myofibroblast differentiation during fibrosis: role of NAD(P)H oxidases

4. The NOX Family of ROS-Generating NADPH Oxidases: Physiology and Pathophysiology

5. Wild-type and mutant p53 differentially regulate NADPH oxidase 4 in TGF-β-mediated migration of human lung and breast epithelial cells

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