The Nrf2/ARE Pathway as a Potential Therapeutic Target in Neurodegenerative Disease-Reference-Cited by-同舟云学术

The Nrf2/ARE Pathway as a Potential Therapeutic Target in Neurodegenerative Disease

Published:2009-03 Issue:3 Volume:11 Page:497-508
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Calkins Marcus J.¹,Johnson Delinda A.¹²,Townsend Jessica A.³,Vargas Marcelo R.²,Dowell James A.²,Williamson Tracy P.²,Kraft Andrew D.²,Lee Jong-Min²,Li Jiang²,Johnson Jeffrey A.¹²³⁴

Affiliation:

1. Molecular and Environmental Toxicology Center, University of Wisconsin, Madison, Wisconsin.

2. Division of Pharmaceutical Sciences, University of Wisconsin, Madison, Wisconsin.

3. Molecular and Cellular Pharmacology, University of Wisconsin, Madison, Wisconsin.

4. Neuroscience Training Program, University of Wisconsin, Madison, Wisconsin.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2008.2242

Reference73 articles.

1. Heme activates the heme oxygenase-1 gene in renal epithelial cells by stabilizing Nrf2

2. Oxidative stress in neurodegeneration: cause or consequence?

3. A role for astrocytes in motor neuron loss in amyotrophic lateral sclerosis

4. Phosphorylation of Nrf2 at Ser40 by Protein Kinase C in Response to Antioxidants Leads to the Release of Nrf2 from INrf2, but Is Not Required for Nrf2 Stabilization/Accumulation in the Nucleus and Transcriptional Activation of Antioxidant Response Element-mediated NAD(P)H:Quinone Oxidoreductase-1 Gene Expression

5. Bioenergetic and oxidative stress in neurodegenerative diseases

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