Roles for Transcription Factors Sp1, NF-κB, IRF3, and IRF7 in Expression of the Human IFNL4 Gene
Author:
Affiliation:
1. National Institute of Biomedical Genomics, Kalyani, West Bengal, India.
2. Transcription and Disease Laboratory, Molecular Biology and Genetics Unit, Jawaharlal Nehru Centre for Advanced Scientific Research, Bangalore, India.
Publisher
Mary Ann Liebert Inc
Subject
Virology,Molecular Medicine,Immunology
Link
https://www.liebertpub.com/doi/pdf/10.1089/vim.2015.0076
Reference34 articles.
1. Impact and Regulation of Lambda Interferon Response in Human Metapneumovirus Infection
2. IL28B expression depends on a novel TT/-G polymorphism which improves HCV clearance prediction
3. Genetic Variants at the IFNL3 Locus and Their Association with Hepatitis C Virus Infections Reveal Novel Insights into Host-Virus Interactions
4. A Single Nucleotide Polymorphism Associated with Hepatitis C Virus Infections Located in the Distal Region of the IL28B Promoter Influences NF-κB-Mediated Gene Transcription
5. Nuclear Factor κB2 p52 Protein Has a Role in Antiviral Immunity through IκB Kinase ϵ-dependent Induction of Sp1 Protein and Interleukin 15
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