Tumor Necrosis Factor Is Critical for Cytolytic T Cell Activity Against Allospecific Hepatocytes and Splenic Targets in Major Histocompatibility Complex Class I Disparate Graft Versus Host Disease-Reference-Cited by-同舟云学术

Tumor Necrosis Factor Is Critical for Cytolytic T Cell Activity Against Allospecific Hepatocytes and Splenic Targets in Major Histocompatibility Complex Class I Disparate Graft Versus Host Disease

Published:2011-05 Issue:5 Volume:31 Page:423-431
ISSN:1079-9907
Container-title:Journal of Interferon & Cytokine Research
language:en
Short-container-title:Journal of Interferon & Cytokine Research

Author:

Ali Sabina¹,Starwalt Ruth²,Kreck Jake²,Whittington Bonnie²,Brown Geri R.³

Affiliation:

1. Department of Pediatrics, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas.

2. Department of Internal Medicine, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas.

3. Division of Digestive and Liver Diseases, Department of Internal Medicine, Dallas Veterans Affairs Medical Center, Dallas, Texas.

Publisher

Mary Ann Liebert Inc

Subject

Virology,Cell Biology,Immunology

Link

http://www.liebertpub.com/doi/pdf/10.1089/jir.2010.0104

Reference25 articles.

1. IL-12-Independent LIGHT Signaling Enhances MHC Class II Disparate CD4+ T Cell Alloproliferation, IFN-γ Responses, and Intestinal Graft-versus-Host Disease

2. Tumor necrosis factor inhibitor ameliorates murine intestinal graft-versus-host disease

3. Enhancement of MHC class I-stimulated alloresponses by TNF/TNF receptor (TNFR)1 interactions and of MHC class II-stimulated alloresponses by TNF/TNFR2 interactions

4. Adenoviral vectors given intravenously to immunocompromised mice yield stable transduction of the colonic epithelium

5. Hepatocytes Express Abundant Surface Class I MHC and Efficiently Use Transporter Associated with Antigen Processing, Tapasin, and Low Molecular Weight Polypeptide Proteasome Subunit Components of Antigen Processing and Presentation Pathway