Increased Urinary Levels of Pentosidine Measured by a Newly Developed Enzyme-Linked Immunosorbent Assay Are Independently Correlated with Fracture After Fall-Reference-Cited by-同舟云学术

Increased Urinary Levels of Pentosidine Measured by a Newly Developed Enzyme-Linked Immunosorbent Assay Are Independently Correlated with Fracture After Fall

Published:2021-12-01 Issue:6 Volume:24 Page:449-455
ISSN:1549-1684
Container-title:Rejuvenation Research
language:en
Short-container-title:Rejuvenation Research

Author:

Tahara Nobuhiro¹^ORCID,Tahara Atsuko¹,Maeda-Ogata Shoko¹,Yoshimura Hanae¹,Bekki Munehisa¹,Sugiyama Yoichi¹,Honda Akihiro¹,Igata Sachiyo¹,Nishino Yuri²,Matsui Takanori²,Fukami Ako¹,Enomoto Mika¹,Adachi Hisashi¹,Fukumoto Yoshihiro¹,Yamagishi Sho-ichi³

Affiliation:

1. Division of Cardiovascular Medicine, Department of Medicine, Kurume University School of Medicine, Kurume, Japan.

2. Department of Pathophysiology and Therapeutics of Diabetic Vascular Complications, Kurume University School of Medicine, Kurume, Japan.

3. Division of Diabetes, Metabolism, and Endocrinology, Department of Medicine, Showa University School of Medicine, Tokyo, Japan.

Publisher

Mary Ann Liebert Inc

Subject

Geriatrics and Gerontology,Aging

Link

https://www.liebertpub.com/doi/pdf/10.1089/rej.2021.0066

Reference51 articles.

1. Evaluation of tissue accumulation levels of advanced glycation end products by skin autofluorescence: A novel marker of vascular complications in high-risk patients for cardiovascular disease

2. The pecking order of skin Advanced Glycation Endproducts (AGEs) as long-term markers of glycemic damage and risk factors for micro- and subclinical macrovascular disease progression in Type 1 diabetes

3. Role of Advanced Glycation Endproduct (AGE)-Receptor for Advanced Glycation Endproduct (RAGE) Axis in Cardiovascular Disease and Its Therapeutic Intervention

4. Development and Progression of Non-Alcoholic Fatty Liver Disease: The Role of Advanced Glycation End Products

5. Advanced Glycation End Products: Building on the Concept of the “Common Soil” in Metabolic Disease