Lipopolysaccharide Promotes Inflammatory Response via Enhancing IFIT1 Expression in Human Umbilical Vein Endothelial Cells-Reference-Cited by-同舟云学术

Lipopolysaccharide Promotes Inflammatory Response via Enhancing IFIT1 Expression in Human Umbilical Vein Endothelial Cells

Published:2020-07-01 Issue:7 Volume:39 Page:1274-1281
ISSN:1044-5498
Container-title:DNA and Cell Biology
language:en
Short-container-title:DNA and Cell Biology

Author:

Wang Jia-Li¹²,Cai Fen¹³,Liu Xue-Hui¹⁴,Li Li-Min¹,He Xin¹,Hu Xiu-Mei¹,Kang Chun-Min⁵,Bai Huan-Lan¹,Zhang Ru-Yi¹,Wu Chang-Meng¹,Wu Li-Mei⁴,Wang Jia⁶,Zheng Lei¹,Ping Bao-Hong⁷,Hu Yan-Wei¹⁸,Wang Qian¹

Affiliation:

1. Laboratory Medicine Center, Nanfang Hospital, Southern Medical University, Guangzhou, China.

2. Linyi People's Hospital of Shandong Province, Linyi, China.

3. Guangzhou Hospital of Integrated Traditional and West Medicine, Guangzhou, China.

4. Department of Clinical Laboratory, Guangzhou Twelfth People's Hospital, Guangzhou, China.

5. Department of Laboratory Medicine, The Second Affiliated Hospital of Guangzhou University of Chinese Medicine, Guangzhou, China.

6. Rizhao People's Hospital of Shandong Province, Rizhao, China.

7. Department of Hui Qiao, Nanfang Hospital, Southern Medical University, Guangzhou, China.

8. Department of Clinical Laboratory, Guangzhou Women & Children Medical Center, Guangzhou Medical University, Guangzhou, China.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Genetics,Molecular Biology,General Medicine

Link

https://www.liebertpub.com/doi/pdf/10.1089/dna.2020.5454

Reference50 articles.

1. Artemisinin inhibits the proliferation, migration, and inflammatory reaction induced by tumor necrosis factor-α in vascular smooth muscle cells through nuclear factor kappa B pathway

2. Ifit1 Protects Against Lipopolysaccharide and D-galactosamine–Induced Fatal Hepatitis by Inhibiting Activation of the JNK Pathway

3. Therapeutic Role of Rifaximin in Inflammatory Bowel Disease: Clinical Implication of Human Pregnane X Receptor Activation

4. Systematic Discovery of TLR Signaling Components Delineates Viral-Sensing Circuits

5. Cell-Specific IRF-3 Responses Protect against West Nile Virus Infection by Interferon-Dependent and -Independent Mechanisms

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