The Microcirculatory Response to Endotoxemia and Resuscitation Is a Marker of Regional Renal Perfusion, Renal Metabolic Stress, and Tubular Injury

Author:

Gomez Hernando1,Haugaa Håkon23,Escobar Daniel4,Botero Ana M.5,Pool Rachel6,Del Rio-Pertuz Gaspar1,Manrique-Caballero Carlos L.1,Gordon Lisa1,Frank Alicia1,Teboul Jean-Louis7,Zuckerbraun Brian S.8,Pinsky Michael R.1

Affiliation:

1. Department of Critical Care Medicine, Center for Critical Care Nephrology, The CRISMA Center, University of Pittsburgh, Pittsburgh, Pennsylvania, USA.

2. Department of Emergencies and Critical Care, Oslo University Hospital, Institute of Clinical Medicine, University of Oslo, Oslo, Norway.

3. Department of Nursing, Lovisenberg Diaconal University College, Oslo, Norway.

4. Department of Obstetrics and Gynecology, Bronx Care Hospital, Bronx, New York, USA.

5. Department of Obstetrics and Gynecology, Staten Island University, Staten Island, New York, USA.

6. Providence-Sacred Heart Medical Center, Spokane, Washington, USA.

7. Service de Médecine Intensive-Réanimation, Hôpital Bicêtre, AP-HP, Université Paris-Saclay, Le Kremlin-Bicêtre, France.

8. Department of Surgery, University of Pittsburgh, Pittsburgh, Pennsylvania, USA.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

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