The Interplay Between Systemic Inflammation, Oxidative Stress, and Tissue Remodeling in Tuberculosis-Reference-Cited by-同舟云学术

The Interplay Between Systemic Inflammation, Oxidative Stress, and Tissue Remodeling in Tuberculosis

Published:2021-02-20 Issue:6 Volume:34 Page:471-485
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Amaral Eduardo P.¹,Vinhaes Caian L.²³⁴,Oliveira-de-Souza Deivide²³⁴,Nogueira Betania³⁴⁵,Akrami Kevan M.³⁵⁶,Andrade Bruno B.²³⁴⁷⁸

Affiliation:

1. Immunobiology Section, Laboratory of Parasitic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, USA.

2. Laboratório de Inflamação e Biomarcadores, Instituto Gonçalo Moniz, Fundação Oswaldo Cruz, Salvador, Brazil.

3. Multinational Organization Network Sponsoring Translational and Epidemiological Research (MONSTER) Initiative, Salvador, Brazil.

4. Curso de Medicina, Faculdade de Tecnologia e Ciências (FTC), Salvador, Brazil.

5. Faculdade de Medicina, Universidade Federal da Bahia, Salvador, Brazil.

6. Division of Infectious Diseases and Pulmonary Critical Care and Sleep Medicine, Department of Medicine, University of California, San Diego, California, USA.

7. Wellcome Centre for Infectious Disease Research in Africa, Institute of Infectious Disease and Molecular Medicine, University of Cape Town, Cape Town, South Africa.

8. Division of Infectious Diseases, Department of Medicine, Vanderbilt University School of Medicine, Nashville, Tennessee, USA.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2020.8124

Reference139 articles.

1. Interleukin-10 Family and Tuberculosis: An Old Story Renewed

2. Vitamin E Status Is Inversely Associated with Risk of Incident Tuberculosis Disease among Household Contacts

3. N-acetyl-cysteine exhibits potent anti-mycobacterial activity in addition to its known anti-oxidative functions

4. A major role for ferroptosis in Mycobacterium tuberculosis–induced cell death and tissue necrosis

5. Inhibiting Adenosine Receptor Signaling Promotes Accumulation of Effector CD4+ T Cells in the Lung Parenchyma During Severe Tuberculosis

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