Epstein–Barr Virus Lytic Reactivation Activates B Cells Polyclonally and Induces Activation-Induced Cytidine Deaminase Expression: A Mechanism Underlying Autoimmunity and Its Contribution to Graves' Disease-Reference-Cited by-同舟云学术

Epstein–Barr Virus Lytic Reactivation Activates B Cells Polyclonally and Induces Activation-Induced Cytidine Deaminase Expression: A Mechanism Underlying Autoimmunity and Its Contribution to Graves' Disease

Published:2017-04 Issue:3 Volume:30 Page:240-249
ISSN:0882-8245
Container-title:Viral Immunology
language:en
Short-container-title:Viral Immunology

Author:

Nagata Keiko¹,Kumata Keisuke¹,Nakayama Yuji²,Satoh Yukio³,Sugihara Hirotsugu¹,Hara Sayuri¹,Matsushita Michiko¹,Kuwamoto Satoshi¹,Kato Masako¹,Murakami Ichiro⁴,Hayashi Kazuhiko¹

Affiliation:

1. Division of Molecular Pathology, Department of Pathology, Faculty of Medicine, Tottori University, Yonago, Japan.

2. Division of Functional Genomics, Research Center for Bioscience and Technology, Tottori University, Yonago, Japan.

3. Division of Biosignaling, Department of Biomedical Sciences, Faculty of Medicine, Tottori University, Yonago, Japan.

4. Department of Pathology, Kochi University Medical School Medical Course, Nankoku-shi, Japan.

Publisher

Mary Ann Liebert Inc

Subject

Virology,Molecular Medicine,Immunology

Link

https://www.liebertpub.com/doi/pdf/10.1089/vim.2016.0179

Reference30 articles.

1. Association of Seasonal Allergic Rhinitis is High in Graves' Disease and Low in Painless Thyroiditis

2. Epstein–Barr Virus Infection

3. EBV-Encoded Latent Membrane Protein 1 Cooperates with BAFF/BLyS and APRIL to Induce T Cell-Independent Ig Heavy Chain Class Switching

4. Epstein-barr virus specific diagnostic tests in infectous mononucleosis

5. Recurrence of thyrotoxicosis after attack of allergic rhinitis in patients with Graves' disease

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