Inhibition of Matrix Metalloproteinases by Lung TIMP-1 Gene Transfer Limits Monocrotaline-Induced Pulmonary Vascular Remodeling in Rats-Reference-Cited by-同舟云学术

Inhibition of Matrix Metalloproteinases by Lung TIMP-1 Gene Transfer Limits Monocrotaline-Induced Pulmonary Vascular Remodeling in Rats

Published:2003-06-10 Issue:9 Volume:14 Page:861-869
ISSN:1043-0342
Container-title:Human Gene Therapy
language:en
Short-container-title:Human Gene Therapy

Author:

Vieillard-Baron Antoine¹,Frisdal Eric²,Raffestin Bernadette³,Baker Andrew H.⁴,Eddahibi Saadia²,Adnot Serge⁵,d'Ortho Marie-Pia⁵

Affiliation:

1. INSERM U492, Faculté de Médecine, 94010 Créteil, France; Service de Réanimation Médicale, Hôpital Ambroise Paré, 92104 Boulogne, France

2. INSERM U492, Faculté de Médecine, 94010 Créteil, France

3. INSERM U492, Faculté de Médecine, 94010 Créteil, France; and Service des Explorations Fonctionnelles, Hôpital Ambroise Paré, 92104 Boulogne, France

4. Department of Medicine and Therapeutics, University of Glasgow, G14ORP Glasgow, United Kingdom

5. INSERM U492, Faculté de Médecine, 94010 Créteil, France; and Service de Physiologie-Explorations Fonctionnelles, Hôpital Henri Mondor, Créteil, France

Publisher

Mary Ann Liebert Inc

Subject

Genetics,Molecular Biology,Molecular Medicine

Link

http://www.liebertpub.com/doi/pdf/10.1089/104303403765701150

Reference34 articles.

1. Development of recombinant adenoviruses that drive high level expression of the human metalloproteinase-9 and tissue inhibitor of metalloproteinase-1 and -2 genes: Characterization of their infection into rabbit smooth muscle cells and human MCF-7 adenocarcinoma cells

2. Divergent effects of tissue inhibitor of metalloproteinase-1, -2, or -3 overexpression on rat vascular smooth muscle cell invasion, proliferation, and death in vitro. TIMP-3 promotes apoptosis.

3. Smooth muscle cell migration and matrix metalloproteinase expression after arterial injury in the rat.

4. Complete reversal of fatal pulmonary hypertension in rats by a serine elastase inhibitor

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