Preclinical Evaluation of Inducible Nitric Oxide Synthase Lipoplex Gene Therapy for Inhibition of Stent-Induced Vascular Neointimal Lesion Formation-Reference-Cited by-同舟云学术

Preclinical Evaluation of Inducible Nitric Oxide Synthase Lipoplex Gene Therapy for Inhibition of Stent-Induced Vascular Neointimal Lesion Formation

Published:2003-03 Issue:4 Volume:14 Page:375-383
ISSN:1043-0342
Container-title:Human Gene Therapy
language:en
Short-container-title:Human Gene Therapy

Author:

Muhs Andreas¹,Heublein Bernd²,Schletter Jens¹,Herrmann Andreas¹,Rüdiger Manfred¹,Sturm Matthias³,Grust Andreas⁴,Malms Jochen⁵,Schrader Jürgen⁶,von der Leyen Heiko E.⁷

Affiliation:

1. Cardion, 40699 Erkrath, Germany

2. Leibniz Institute for Biotechnology and Artificial Organs (LEBAO), 30559 Hannover, Germany

3. Abteilung für Kardiologie, Medizinische Hochschule Hannover, 30623 Hannover, Germany

4. Institut für Diagnostische Radiologie, Heinrich-Heine-Universität Düsseldorf, 40225 Düsseldorf, Germany

5. Institut für Diagnostische Radiologie, Allgemeines Krankenhaus Viersen, 41747 Viersen, Germany

6. Institut für Herz und Kreislaufphysiologie, Heinrich-Heine-Universität Düsseldorf, 40225 Düsseldorf, Germany

7. Cardion, 40699 Erkrath, Germany; Leibniz Institute for Biotechnology and Artificial Organs (LEBAO), 30559 Hannover, Germany

Publisher

Mary Ann Liebert Inc

Subject

Genetics,Molecular Biology,Molecular Medicine

Link

http://www.liebertpub.com/doi/pdf/10.1089/104303403321208970

Reference24 articles.

1. A novel role for the cyclin-dependent kinase inhibitor p27Kip1 in angiotensin II–stimulated vascular smooth muscle cell hypertrophy

2. Morphologic characteristics of lesion formation and time course of smooth muscle cell proliferation in a porcine proliferative restenosis model

3. Induction of nitric oxide synthase in human vascular smooth muscle: interactions between proinflammatory cytokines

4. Nitric oxide inhibits angiotensin II-induced migration of rat aortic smooth muscle cell. Role of cyclic-nucleotides and angiotensin1 receptors.

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