The MAVS Immune Recognition Pathway in Viral Infection and Sepsis-Reference-Cited by-同舟云学术

The MAVS Immune Recognition Pathway in Viral Infection and Sepsis

Published:2021-12-01 Issue:16 Volume:35 Page:1376-1392
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Sharma Arjun¹²^ORCID,Kontodimas Konstantinos¹,Bosmann Markus¹²^ORCID

Affiliation:

1. Pulmonary Center, Department of Medicine, Boston University School of Medicine, Boston, Massachusetts, USA.

2. Center for Thrombosis and Hemostasis, University Medical Center of the Johannes Gutenberg-University, Mainz, Germany.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2021.0167

Reference170 articles.

1. Influence of Systemic Inflammatory Response Syndrome and Sepsis on Outcome of Critically Ill Infected Patients

2. Neutrophil extracellular traps impair fungal clearance in a mouse model of invasive pulmonary aspergillosis

3. Cleavage of mitochondrial antiviral signaling protein in the liver of patients with chronic hepatitis C correlates with a reduced activation of the endogenous interferon system

4. The global distribution and burden of dengue

5. MAVS and MyD88 are essential for innate immunity but not cytotoxic T lymphocyte response against respiratory syncytial virus

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