RORα Decreases Oxidative Stress Through the Induction of SOD2 and GPx1 Expression and Thereby Protects Against Nonalcoholic Steatohepatitis in Mice-Reference-Cited by-同舟云学术

RORα Decreases Oxidative Stress Through the Induction of SOD2 and GPx1 Expression and Thereby Protects Against Nonalcoholic Steatohepatitis in Mice

Published:2014-11-20 Issue:15 Volume:21 Page:2083-2094
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Han Yong-Hyun¹,Kim Hyeon-Ji¹,Kim Eun-Jin¹,Kim Kyu-Seo¹,Hong Suckchang¹,Park Hyeung-Geun¹,Lee Mi-Ock¹

Affiliation:

1. Bio-MAX Institute, Research Institute of Pharmaceutical Sciences, College of Pharmacy, Seoul National University, Seoul, Korea.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2013.5655

Reference43 articles.

1. Role of Alcohol Metabolism in Non-Alcoholic Steatohepatitis

2. Human retinoic acid receptor-related orphan receptor ?1 overexpression protects neurones against oxidative stress-induced apoptosis

3. Expanding the natural history of nonalcoholic steatohepatitis: From cryptogenic cirrhosis to hepatocellular carcinoma

4. The MIQE Guidelines: Minimum Information for Publication of Quantitative Real-Time PCR Experiments

5. Favorable effects of a prolonged treatment with melatonin on the level of oxidative damage and neurodegeneration in senescence-accelerated mice

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