HypervirulentM. tuberculosisW/Beijing Strains Upregulate Type I IFNs and Increase Expression of Negative Regulators of the Jak-Stat Pathway-Reference-Cited by-同舟云学术

HypervirulentM. tuberculosisW/Beijing Strains Upregulate Type I IFNs and Increase Expression of Negative Regulators of the Jak-Stat Pathway

Published:2005-11 Issue:11 Volume:25 Page:694-701
ISSN:1079-9907
Container-title:Journal of Interferon & Cytokine Research
language:en
Short-container-title:Journal of Interferon & Cytokine Research

Author:

Manca Claudia¹,Tsenova Liana¹,Freeman Sherry¹,Barczak Amy K.²,Tovey Michael³,Murray Peter J.⁴,Barry Clifton²,Kaplan Gilla¹

Affiliation:

1. Laboratory of Mycobacterial Immunity and Pathogenesis, Public Health Research Institute at the International Center for Public Health, Newark, NJ 07103-3535.

2. Tuberculosis Research Section, NIAID, National Institutes of Health, Rockville, MD 20852.

3. Laboratory of Viral Oncology UPR 9045 C.N.R.S, Institute Andre Lwoff, 94801 Villejuif, France.

4. Department of Infectious Diseases, St. Jude Children's Research Hospital, Memphis, TN 38105.

Publisher

Mary Ann Liebert Inc

Subject

Virology,Cell Biology,Immunology

Link

http://www.liebertpub.com/doi/pdf/10.1089/jir.2005.25.694

Reference36 articles.

1. Interaction of antigen presenting cells with mycobacteria

2. Alternate Class I MHC Antigen Processing Is Inhibited by Toll-Like Receptor Signaling Pathogen-Associated Molecular Patterns:Mycobacterium tuberculosis19-kDa Lipoprotein, CpG DNA, and Lipopolysaccharide

3. Restraining mycobacteria: Role of granulomas in mycobacterial infections

4. An essential role for interferon gamma in resistance to Mycobacterium tuberculosis infection.

5. Interferon-gamma-treated murine macrophages inhibit growth of tubercle bacilli via the generation of reactive nitrogen intermediates

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