The Cystine/Glutamate Antiporter System xc−in Health and Disease: From Molecular Mechanisms to Novel Therapeutic Opportunities-Reference-Cited by-同舟云学术

The Cystine/Glutamate Antiporter System xc−in Health and Disease: From Molecular Mechanisms to Novel Therapeutic Opportunities

Published:2013-02-10 Issue:5 Volume:18 Page:522-555
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Lewerenz Jan¹,Hewett Sandra J.²,Huang Ying³,Lambros Maria³,Gout Peter W.⁴,Kalivas Peter W.⁵,Massie Ann⁶,Smolders Ilse⁷,Methner Axel⁸,Pergande Mathias⁸,Smith Sylvia B.⁹,Ganapathy Vadivel¹⁰,Maher Pamela¹¹

Affiliation:

1. Department of Neurology, University of Ulm, Ulm, Germany.

2. Program in Neuroscience, Department of Biology, Syracuse University, Syracuse, New York.

3. Center for the Advancement of Drug Research and Evaluation, College of Pharmacy, Western University of Health Sciences, Pomona, California.

4. Department of Experimental Therapeutics, BC Cancer Agency, Vancouver, British Columbia, Canada.

5. Department of Neurosciences, Medical University of South Carolina, Charleston, South Carolina.

6. Department of Pharmaceutical Biotechnology and Molecular Biology, Vrije Universiteit Brussel, Brussels, Belgium.

7. Department of Pharmaceutical Chemistry and Drug Analysis, Center for Neuroscience, Vrije Universiteit Brussel, Brussels, Belgium.

8. Department of Neurology, Heinrich-Heine-University Duesseldorf, Duesseldorf, Germany.

9. Department of Cellular Biology and Anatomy, Medical College of Georgia, Augusta, Georgia.

10. Department of Biochemistry and Molecular Biology, Medical College of Georgia, Augusta, Georgia.

11. Cellular Neurobiology Laboratory, Salk Institute for Biological Studies, La Jolla, California.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2011.4391

Reference300 articles.

1. ROS-Production–Mediated Activation of AP-1 but Not NFκB Inhibits Glutamate-Induced HT4 Neuronal Cell Death

2. Mechanisms of Oxidative Glutamate Toxicity: The Glutamate/Cystine Antiporter System xc¯ as a Neuroprotective Drug Target

3. Control of extracellular cysteine/cystine redox state by HT-29 cells is independent of cellular glutathione

4. The early phase of critical illness is a progressive acidic state due to unmeasured anions

5. Neuroprotective effects of zonisamide target astrocyte

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