Redox Proteomics Analyses of the Influence of Co-Expression of Wild-Type or Mutated LRRK2 and Tau on C. elegans Protein Expression and Oxidative Modification: Relevance to Parkinson Disease-Reference-Cited by-同舟云学术

Redox Proteomics Analyses of the Influence of Co-Expression of Wild-Type or Mutated LRRK2 and Tau on C. elegans Protein Expression and Oxidative Modification: Relevance to Parkinson Disease

Published:2012-12 Issue:11 Volume:17 Page:1490-1506
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Di Domenico Fabio¹²,Sultana Rukhsana¹,Ferree Andrew³,Smith Katelyn³,Barone Eugenio¹⁴,Perluigi Marzia²,Coccia Raffaella²,Pierce William⁵,Cai Jian⁵,Mancuso Cesare⁴,Squillace Rachel⁶,Wiengele Manfred⁶,Dalle-Donne Isabella⁷,Wolozin Benjamin³⁸,Butterfield D. Allan²

Affiliation:

1. Department of Chemistry, Center of Membrane Sciences, and Sanders-Brown Center on Aging, University of Kentucky, Lexington, Kentucky.

2. Department of Biochemical Sciences, Sapienza University of Rome, Rome, Italy.

3. Department of Pharmacology, Boston University, Boston, Massachusetts.

4. Institute of Pharmacology, Catholic University of Rome, Rome, Italy.

5. Department of Pharmacology, University of Louisville, Louisville, Kentucky.

6. Ariad Pharmaceuticals, Cambridge, Massachusetts.

7. Department of Biology, University of Milan, Milan, Italy.

8. Edith Nourse Rogers Memorial Veterans Affairs Medical Center, Bedford, Massachusetts.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2011.4312

Reference61 articles.

1. Expanding insights of mitochondrial dysfunction in Parkinson's disease

2. LRRK2 regulates autophagic activity and localizes to specific membrane microdomains in a novel human genomic reporter cellular model

3. Parkinson disease, LRRK2 and the endocytic-autophagic pathway

4. Redox Proteomics Analysis of Brains from Subjects with Amnestic Mild Cognitive Impairment Compared to Brains from Subjects with Preclinical Alzheimer's Disease: Insights into Memory Loss in MCI

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