p62 at the Interface of Autophagy, Oxidative Stress Signaling, and Cancer-Reference-Cited by-同舟云学术

p62 at the Interface of Autophagy, Oxidative Stress Signaling, and Cancer

Published:2012-09 Issue:5 Volume:17 Page:786-793
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Nezis Ioannis P.¹²,Stenmark Harald¹²

Affiliation:

1. Centre for Cancer Biomedicine, Faculty of Medicine, University of Oslo, Oslo, Norway.

2. Department of Biochemistry, Institute for Cancer Research, Oslo University Hospital, Oslo, Norway.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2011.4394

Reference46 articles.

1. The Drosophila Atypical Protein Kinase C-Ref(2)P Complex Constitutes a Conserved Module for Signaling in the Toll Pathway

2. p62/SQSTM1 forms protein aggregates degraded by autophagy and has a protective effect on huntingtin-induced cell death

3. Control of Sigma Virus Multiplication by the ref(2)P Gene of Drosophila melanogaster: An in Vivo Study of the PB1 Domain of Ref(2)P

4. Physical and Functional Interaction of Sequestosome 1 with Keap1 Regulates the Keap1-Nrf2 Cell Defense Pathway

5. Oncogene-induced Nrf2 transcription promotes ROS detoxification and tumorigenesis

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