Loss of Tenomodulin Results in Reduced Self-Renewal and Augmented Senescence of Tendon Stem/Progenitor Cells-Reference-Cited by-同舟云学术

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Loss of Tenomodulin Results in Reduced Self-Renewal and Augmented Senescence of Tendon Stem/Progenitor Cells

Published:2015-03 Issue:5 Volume:24 Page:597-609
ISSN:1547-3287
Container-title:Stem Cells and Development
language:en
Short-container-title:Stem Cells and Development

Author:

Alberton Paolo¹,Dex Sarah¹,Popov Cvetan¹,Shukunami Chisa²³,Schieker Matthias¹,Docheva Denitsa¹

Affiliation:

1. Experimental Surgery and Regenerative Medicine, Department of Surgery, Ludwig-Maximilians-University (LMU), Munich, Germany.

2. Department of Cellular Differentiation, Institute for Frontier Medical Sciences, Kyoto University, Kyoto, Japan.

3. Department of Molecular Biology and Biochemistry, Institute of Biomedical and Health Sciences, Hiroshima, Japan.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Developmental Biology,Hematology

Link

https://www.liebertpub.com/doi/pdf/10.1089/scd.2014.0314

Reference38 articles.

1. A novel gene,tendin, is strongly expressed in tendons and ligaments and shows high homology with chondromodulin-I

2. Molecular Cloning of tenomodulin, a Novel Chondromodulin-I Related Gene

3. Tenomodulin Is Necessary for Tenocyte Proliferation and Tendon Maturation

4. Local Tenomodulin Absence, Angiogenesis, and Matrix Metalloproteinase Activation Are Associated With the Rupture of the Chordae Tendineae Cordis

5. Direct conversion of tenocytes into chondrocytes by Sox9

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