Persistent Neuroinflammation May Be Involved in the Pathogenesis of Traumatic Brain Injury (TBI)-Induced Hypopituitarism: Potential Genetic and Autoimmune Factors-Reference-Cited by-同舟云学术

Persistent Neuroinflammation May Be Involved in the Pathogenesis of Traumatic Brain Injury (TBI)-Induced Hypopituitarism: Potential Genetic and Autoimmune Factors

Published:2010-02 Issue:2 Volume:27 Page:301-302
ISSN:0897-7151
Container-title:Journal of Neurotrauma
language:en
Short-container-title:Journal of Neurotrauma

Author:

Tanriverdi Fatih¹,Unluhizarci Kursad¹,Kelestrimur Fahrettin¹

Affiliation:

1. Department of Endocrinology, Erciyes University Medical School, Kayseri, Turkey.

Publisher

Mary Ann Liebert Inc

Subject

Neurology (clinical)

Link

http://www.liebertpub.com/doi/pdf/10.1089/neu.2009.1102

Reference7 articles.

1. Apolipoprotein E suppresses glial cell secretion of TNFα

2. Downregulation of Microglial Activation by Apolipoprotein E and ApoE-Mimetic Peptides

3. A novel therapeutic derived from apolipoprotein E reduces brain inflammation and improves outcome after closed head injury

4. The top 10 most abundant transcripts are sufficient to characterize the organs functional specificity: evidences from the cortex, hypothalamus and pituitary gland

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