Hypoxia Activates NF-κB–Dependent Gene Expression Through the Canonical Signaling Pathway-Reference-Cited by-同舟云学术

Hypoxia Activates NF-κB–Dependent Gene Expression Through the Canonical Signaling Pathway

Published:2009-09 Issue:9 Volume:11 Page:2057-2064
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Oliver Kathryn M.¹,Garvey John F.¹,Ng Chin Teck²,Veale Douglas J.¹²,Fearon Ursula¹²,Cummins Eoin P.¹³,Taylor Cormac T.¹³

Affiliation:

1. UCD Conway Institute, University College Dublin, Belfield, Ireland.

2. Education, and Research Centre, St. Vincent's University Hospital, Dublin, Ireland.

3. These authors contributed equally to this study.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2008.2400

Reference27 articles.

1. Posttranslational hydroxylation of ankyrin repeats in I B proteins by the hypoxia-inducible factor (HIF) asparaginyl hydroxylase, factor inhibiting HIF (FIH)

2. Prolyl hydroxylase-1 negatively regulates I B kinase-beta, giving insight into hypoxia-induced NF B activity

3. Hypoxia-responsive transcription factors

4. The alternative NF-κB pathway from biochemistry to biology: Pitfalls and promises for future drug development

5. RelB/p50 Dimers Are Differentially Regulated by Tumor Necrosis Factor-α and Lymphotoxin-β Receptor Activation

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