Thrombospondin-1, Free Radicals, and the Coronary Microcirculation: The Aging Conundrum-Reference-Cited by-同舟云学术

Thrombospondin-1, Free Radicals, and the Coronary Microcirculation: The Aging Conundrum

Published:2017-10-20 Issue:12 Volume:27 Page:785-801
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

LeBlanc Amanda J.¹,Kelm Natia Q.¹

Affiliation:

1. Department of Physiology, Cardiovascular Innovation Institute, University of Louisville, Louisville, Kentucky.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2017.7292

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1. NAD(P)H oxidase-generated superoxide anion accounts for reduced control of myocardial O2 consumption by NO in old Fischer 344 rats

2. Adipose-derived stromal vascular fraction cells isolated from old animals exhibit reduced capacity to support the formation of microvascular networks

3. Expression of CD47 (integrin-associated protein) decreases on red blood cells during storage

4. MMP-9-Dependent Serum-Borne Bioactivity Caused by Multiwalled Carbon Nanotube Exposure Induces Vascular Dysfunction via the CD36 Scavenger Receptor

5. Serum-borne bioactivity caused by pulmonary multiwalled carbon nanotubes induces neuroinflammation via blood–brain barrier impairment

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