The Extracellular Domain of p185c-neuInduces Density-Dependent Inhibition of Cell Growth in Malignant Mesothelioma Cells and Reduces Growth of MesotheliomaIn Vivo-Reference-Cited by-同舟云学术

The Extracellular Domain of p185c-neuInduces Density-Dependent Inhibition of Cell Growth in Malignant Mesothelioma Cells and Reduces Growth of MesotheliomaIn Vivo

Published:2006-09 Issue:9 Volume:25 Page:530-540
ISSN:1044-5498
Container-title:DNA and Cell Biology
language:en
Short-container-title:DNA and Cell Biology

Author:

Yoneda Tsutomu¹,Kumagai Toru¹,Nagatomo Izumi¹,Furukawa Mitsugi¹,Yamane Hiroyuki¹,Hoshino Shigenori¹,Mori Masahide¹,Takeda Yoshito¹,Horai Takeshi²,Nishida Sumiyuki¹,Watanabe Dai¹,Kijima Takashi¹,Yoshida Mitsuhiro¹,Osaki Tadashi¹,Tachibana Isao¹,Greene Mark I.³,Kawase Ichiro¹

Affiliation:

1. Department of Respiratory Medicine, Allergy and Rheumatic Diseases, Osaka University Graduate School of Medicine, Osaka, Japan.

2. Cancer Institute Hospital, Tokyo, Japan.

3. Department of Pathology and Laboratory Medicine, University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Genetics,Molecular Biology,General Medicine

Link

http://www.liebertpub.com/doi/pdf/10.1089/dna.2006.25.530

Reference62 articles.

1. Expression of a truncated 100 kDa HER2 splice variant acts as an endogenous inhibitor of tumour cell proliferation

2. Expression of herstatin, an autoinhibitor of HER-2/neu, inhibits transactivation of HER-3 by HER-2 and blocks EGF activation of the EGF receptor

3. The neu oncogene encodes an epidermal growth factor receptor-related protein

4. p27 immunostaining is related to prognosis in malignant mesothelioma

5. Disabling Receptor Ensembles with Rationally Designed Interface Peptidomimetics

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