Endoglin Level Is Critical for Cartilage Tissue Formation In Vitro by Passaged Human Chondrocytes-Reference-Cited by-同舟云学术

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Endoglin Level Is Critical for Cartilage Tissue Formation In Vitro by Passaged Human Chondrocytes

Published:2021-09-01 Issue:17-18 Volume:27 Page:1140-1150
ISSN:1937-3341
Container-title:Tissue Engineering Part A
language:en
Short-container-title:Tissue Engineering Part A

Author:

Bianchi Vanessa J.¹²,Parsons Michael¹,Backstein David³,Kandel Rita A.¹²⁴⁵

Affiliation:

1. Lunenfeld-Tanenbaum Research Institute, Toronto, Canada.

2. Institute of Biomaterials and Biomedical Engineering, University of Toronto, Toronto, Canada.

3. Division of Orthopaedic Surgery, Mount Sinai Hospital, Toronto, Canada.

4. Department of Pathology and Laboratory Medicine, Mount Sinai Hospital, Toronto, Canada.

5. Department of Laboratory Medicine and Pathobiology, University of Toronto, Toronto, Canada.

Publisher

Mary Ann Liebert Inc

Subject

Biomedical Engineering,Biomaterials,Biochemistry,Bioengineering

Link

https://www.liebertpub.com/doi/pdf/10.1089/ten.tea.2020.0120

Reference49 articles.

1. The global burden of hip and knee osteoarthritis: estimates from the Global Burden of Disease 2010 study

2. The interleukin-1 receptor in normal and osteoarthritic human articular chondrocytes. Identification as the type I receptor and analysis of binding kinetics and biologic function

3. Chondrocyte tumor necrosis factor receptors and focal loss of cartilage in osteoarthritis

4. Freshly isolated osteoarthritic chondrocytes are catabolically more active than normal chondrocytes, but less responsive to catabolic stimulation with interleukin-1?

5. Phenotypic modulation of chondrocytes as a potential therapeutic target in osteoarthritis: a hypothesis

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The roles and regulatory mechanisms of TGF-β and BMP signaling in bone and cartilage development, homeostasis and disease;Cell Research;2024-01-24

2. Endoglin aggravates peritoneal fibrosis by regulating the activation of TGF-β/ALK/Smads signaling;Frontiers in Pharmacology;2022-09-23

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