The 4th and 112th Residues of Viral Capsid Cooperatively Modulate Capsid-CPSF6 Interactions of HIV-1-Reference-Cited by-同舟云学术

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The 4th and 112th Residues of Viral Capsid Cooperatively Modulate Capsid-CPSF6 Interactions of HIV-1

Published:2020-06-01 Issue:6 Volume:36 Page:513-521
ISSN:0889-2229
Container-title:AIDS Research and Human Retroviruses
language:en
Short-container-title:AIDS Research and Human Retroviruses

Author:

Saito Akatsuki¹,Sultana Tahmina¹,Ode Hirotaka²,Nohata Kyotaro¹,Samune Yoshihiro¹,Nakayama Emi E.¹,Iwatani Yasumasa²³,Shioda Tatsuo¹

Affiliation:

1. Research Institute for Microbial Diseases, Osaka University, Osaka, Japan.

2. Clinical Research Center, National Hospital Organization Nagoya Medical Center, Nagoya, Japan.

3. Division of Basic Medicine, Nagoya University Graduate School of Medicine, Nagoya, Japan.

Publisher

Mary Ann Liebert Inc

Subject

Infectious Diseases,Virology,Immunology

Link

https://www.liebertpub.com/doi/pdf/10.1089/aid.2019.0250

Reference28 articles.

1. A critical role for alternative polyadenylation factor CPSF6 in targeting HIV-1 integration to transcriptionally active chromatin

2. Capsid-CPSF6 Interaction Is Dispensable for HIV-1 Replication in Primary Cells but Is Selected during Virus Passage In Vivo

3. HIV-1 Resistance to the Capsid-Targeting Inhibitor PF74 Results in Altered Dependence on Host Factors Required for Virus Nuclear Entry

4. Flexible Use of Nuclear Import Pathways by HIV-1

5. CPSF6 Defines a Conserved Capsid Interface that Modulates HIV-1 Replication

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Human immunodeficiency virus-1 core: The Trojan horse in virus–host interaction;Frontiers in Microbiology;2022-08-29

2. HIV-1 capsid variability: viral exploitation and evasion of capsid-binding molecules;Retrovirology;2021-10-26

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