Increased Expression of p22phox Mediates Airway Hyperresponsiveness in an Experimental Model of Asthma-Reference-Cited by-同舟云学术

Increased Expression of p22phox Mediates Airway Hyperresponsiveness in an Experimental Model of Asthma

Published:2017-12-20 Issue:18 Volume:27 Page:1460-1472
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Nagaraj Chandran¹,Haitchi Hans Michael²³⁴,Heinemann Akos⁵,Howarth Peter H.³,Olschewski Andrea¹⁶,Marsh Leigh M.¹

Affiliation:

1. Ludwig Boltzmann Institute for Lung Vascular Research, Graz, Austria.

2. The Brooke Laboratory, Academic Unit of Clinical and Experimental Sciences, Faculty of Medicine, University of Southampton, Southampton, United Kingdom.

3. National Institute for Health Research (NIHR) Southampton Respiratory Biomedical Research Unit, University Hospital Southampton NHS Foundation Trust, Southampton, United Kingdom.

4. Institute for Life Sciences, University of Southampton, Southampton, United Kingdom.

5. Institute of Experimental and Clinical Pharmacology, Medical University of Graz, Graz, Austria.

6. Department of Physiology, Medical University of Graz, Graz, Austria.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2016.6863

Reference47 articles.

1. Compartmental and Temporal Dynamics of Chronic Inflammation and Airway Remodelling in a Chronic Asthma Mouse Model

2. Direct Interaction of the Novel Nox Proteins with p22phox Is Required for the Formation of a Functionally Active NADPH Oxidase

3. Defining the molecular role of gp91phox in the immune manifestation of acute allergic asthma using a preclinical murine model

4. Role of T cells in a gp91phox knockout murine model of acute allergic asthma

5. Reactive oxygen species and airway inflammation

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