Heme Causes Pain in Sickle Mice via Toll-Like Receptor 4-Mediated Reactive Oxygen Species- and Endoplasmic Reticulum Stress-Induced Glial Activation-Reference-Cited by-同舟云学术

Heme Causes Pain in Sickle Mice via Toll-Like Receptor 4-Mediated Reactive Oxygen Species- and Endoplasmic Reticulum Stress-Induced Glial Activation

Published:2021-02-01 Issue:4 Volume:34 Page:279-293
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Lei Jianxun¹,Paul Jinny¹,Wang Ying¹,Gupta Mihir²,Vang Derek¹,Thompson Susan¹,Jha Ritu¹,Nguyen Julia¹,Valverde Yessenia¹,Lamarre Yann¹,Jones Michael K.³⁴,Gupta Kalpna¹³⁴

Affiliation:

1. Vascular Biology Center, Division of Hematology, Oncology & Transplantation, Department of Medicine, University of Minnesota, Minneapolis, Minnesota, USA.

2. Department of Neurosurgery, University of California San Diego, La Jolla, California, USA.

3. Division of Hematology/Oncology, Department of Medicine, University of California, Irvine, Irvine, California, USA.

4. Southern California Institute for Research and Education, Long Beach, California, USA.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2019.7913

Reference66 articles.

1. Association between plasma free haem and incidence of vaso-occlusive episodes and acute chest syndrome in children with sickle cell disease

2. Protease-activated receptor 2 sensitizes TRPV1 by protein kinase Cɛ- and A-dependent mechanisms in rats and mice

3. Transgenic sickle mice have vascular inflammation

4. Heme triggers TLR4 signaling leading to endothelial cell activation and vaso-occlusion in murine sickle cell disease

5. A Selective Inhibitor of eIF2α Dephosphorylation Protects Cells from ER Stress

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