Nitric Oxide-Dependent Protein Post-Translational Modifications Impair Mitochondrial Function and Metabolism to Contribute to Neurodegenerative Diseases-Reference-Cited by-同舟云学术

Nitric Oxide-Dependent Protein Post-Translational Modifications Impair Mitochondrial Function and Metabolism to Contribute to Neurodegenerative Diseases

Published:2020-04-20 Issue:12 Volume:32 Page:817-833
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Nakamura Tomohiro¹,Lipton Stuart A.¹²

Affiliation:

1. Departments of Molecular Medicine and Neuroscience, Neuroscience Translational Center, The Scripps Research Institute, La Jolla, California.

2. Department of Neurosciences, University of California San Diego, School of Medicine, La Jolla, California.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2019.7916

Reference153 articles.

1. Cysteinyl-tRNA synthetase governs cysteine polysulfidation and mitochondrial bioenergetics

2. Identification of S-nitroso-CoA reductases that regulate protein S-nitrosylation

3. Oxidative stress in neurodegeneration: cause or consequence?

4. Mechanisms of Reduced Striatal NMDA Excitotoxicity in Type I Nitric Oxide Synthase Knock-Out Mice

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