Prolonged Interferon-Stimulated Gene and Protein Signatures in Multiple Sclerosis Induced by PEGylated IFN-β-1a Compared to Non-PEGylated IFN-β-1a
Author:
Affiliation:
1. Department of Neurology, Neurology A-205, MC-2030, University of Chicago Medicine, Chicago, Illinois, USA.
Publisher
Mary Ann Liebert Inc
Subject
Virology,Cell Biology,Immunology
Link
https://www.liebertpub.com/doi/pdf/10.1089/jir.2022.0238
Reference31 articles.
1. CDK8 Kinase Phosphorylates Transcription Factor STAT1 to Selectively Regulate the Interferon Response
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4. Immunopathology of multiple sclerosis
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1. Efficacy and safety of peginterferon beta-1a compared to interferon beta-1a in relapsing remitting multiple sclerosis patients: A phase 3, randomized, non-inferiority clinical trial (PEGINTEGRITY);Multiple Sclerosis and Related Disorders;2024-10
2. Alternative Splicing in Multiple Sclerosis: A Promising Biomarker of Therapeutic Response to Interferon-β;Journal of Interferon & Cytokine Research;2024-08-01
3. Alternative Splicing of RNA Is Excessive in Multiple Sclerosis and Not Linked to Gene Expression Levels: Dysregulation Is Corrected by IFN-β;Journal of Interferon & Cytokine Research;2024-08-01
4. Novel biomarkers and interferon signature in secondary progressive multiple sclerosis;Journal of Neuroimmunology;2024-04
5. Darren P. Baker, PhD A Pioneer in the Development of Pegylated Interferon-β for Clinical Use;Journal of Interferon & Cytokine Research;2023-03-01
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