Potential of Radiation-Induced Cellular Stress for Reactivation of Latent HIV-1 and Killing of Infected Cells
Author:
Affiliation:
1. National Center for Biodefense and Infectious Diseases, George Mason University, Manassas, Virginia.
2. Laboratory of Molecular Virology, School of Systems Biology, George Mason University, Manassas, Virginia.
Publisher
Mary Ann Liebert Inc
Subject
Infectious Diseases,Virology,Immunology
Link
http://www.liebertpub.com/doi/pdf/10.1089/aid.2016.0006
Reference62 articles.
1. Replication-Competent Noninduced Proviruses in the Latent Reservoir Increase Barrier to HIV-1 Cure
2. Disulfiram Reactivates Latent HIV-1 in a Bcl-2-Transduced Primary CD4 + T Cell Model without Inducing Global T Cell Activation
3. Expression of Latent HIV Induced by the Potent HDAC Inhibitor Suberoylanilide Hydroxamic Acid
4. Administration of vorinostat disrupts HIV-1 latency in patients on antiretroviral therapy
5. Ex vivo analysis identifies effective HIV-1 latency–reversing drug combinations
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