Soluble Factors Secreted by Endothelial Cells Allow for Productive and Latent HIV-1 Infection in Resting CD4+T Cells
Author:
Affiliation:
1. Department of Biology, Calvin College, Grand Rapids, Michigan.
2. Department of Pathology and Laboratory Medicine, University of Pennsylvania, Philadelphia, Pennsylvania.
Publisher
Mary Ann Liebert Inc
Subject
Infectious Diseases,Virology,Immunology
Link
http://www.liebertpub.com/doi/pdf/10.1089/aid.2016.0058
Reference33 articles.
1. Reservoirs for HIV-1: Mechanisms for Viral Persistence in the Presence of Antiviral Immune Responses and Antiretroviral Therapy
2. Resistant viral variants in cellular reservoirs of human immunodeficiency virus infection
3. Human Immunodeficiency Virus (HIV)-1 Integration Sites in Viral Latency
4. An In-Depth Comparison of Latent HIV-1 Reactivation in Multiple Cell Model Systems and Resting CD4+ T Cells from Aviremic Patients
5. Progression to the G 1 b Phase of the Cell Cycle Is Required for Completion of Human Immunodeficiency Virus Type 1 Reverse Transcription in T Cells
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1. Intestinal endothelial cells increase HIV infection and latency in resting and activated CD4 + T cells, particularly affecting CCR6 + CD4 + T cells;Retrovirology;2023-05-18
2. The reservoir of latent HIV;Frontiers in Cellular and Infection Microbiology;2022-07-28
3. Endothelial Cells Promote Productive HIV Infection of Resting CD4+ T Cells by an Integrin-Mediated Cell Adhesion-Dependent Mechanism;AIDS Research and Human Retroviruses;2022-02-01
4. Endothelial cells promote productive HIV infection of resting CD4+ T cells by an integrin-mediated cell adhesion-dependent mechanism;2020-06-29
5. Lymphatic endothelial cells promote productive and latent HIV infection in resting CD4+ T cells;Virology Journal;2018-10-03
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