Lesional Expression of RhoA and RhoB following Traumatic Brain Injury in Humans-Reference-Cited by-同舟云学术

Lesional Expression of RhoA and RhoB following Traumatic Brain Injury in Humans

Published:2004-06 Issue:6 Volume:21 Page:697-706
ISSN:0897-7151
Container-title:Journal of Neurotrauma
language:en
Short-container-title:Journal of Neurotrauma

Author:

Brabeck Christine¹,Beschorner Rudi¹,Conrad Sabine¹,Mittelbronn Michel¹,Bekure Kubrom¹,Meyermann Richard¹,Schluesener Hermann J.¹,Schwab Jan M.²

Affiliation:

1. Institute of Brain Research, University of Tuebingen, Tuebingen, Germany.

2. Institute of Brain Research, University of Tuebingen, Tuebingen, Germany and CNRS UMR 7102, Université Pierre et Marie Curie (Paris 6), Equipe Développement Neuronal, Paris, France.

Publisher

Mary Ann Liebert Inc

Subject

Neurology (clinical)

Link

http://www.liebertpub.com/doi/pdf/10.1089/0897715041269597

Reference45 articles.

1. Activation of RhoA and ROCK Are Essential for Detachment of Migrating Leukocytes

2. Long-term expression of heme oxygenase-1 (HO-1, HSP-32) following focal cerebral infarctions and traumatic brain injury in humans

3. The mammalian G protein rhoC is ADP-ribosylated by Clostridium botulinum exoenzyme C3 and affects actin microfilaments in Vero cells.

4. Genomic analysis of metastasis reveals an essential role for RhoC

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