Nociceptin/Orphanin FQ Alters Prostaglandin Cerebrovascular Action following Brain Injury
Author:
Affiliation:
1. Departments of Anesthesia and Pharmacology, University of Pennsylvania, Philadelphia, Pennsylvania
Publisher
Mary Ann Liebert Inc
Subject
Clinical Neurology
Link
http://www.liebertpub.com/doi/pdf/10.1089/089771504322778640
Reference25 articles.
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4. Role of impaired cAMP and calcium-sensitive K+ channel function in altered cerebral hemodynamics following brain injury
5. Brain Injury Impairs Prostaglandin Cerebrovasodilation
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1. Therapeutic potential of nociceptin/orphanin FQ peptide (NOP) receptor modulators for treatment of traumatic brain injury, traumatic stress, and their co-morbidities;Pharmacology & Therapeutics;2022-03
2. PAI-1-Derived Peptide EEIIMD Prevents Hypoxia/Ischemia-Induced Aggravation of Endothelin- and Thromboxane-Induced Cerebrovasoconstriction;Neurocritical Care;2013-11-19
3. Differential activation of ERK, p38, and JNK MAPK by nociceptin/orphanin FQ in the potentiation of prostaglandin cerebrovasoconstriction after brain injury;European Journal of Pharmacology;2006-01
4. Endogenous opiates and behavior: 2004;Peptides;2005-12
5. Sample-Dependent Effects on the Neuropeptidome Detected in Rat Brain Tissue Preparations by Capillary Liquid Chromatography with Tandem Mass Spectrometry;Analytical Chemistry;2005-09-01
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