Targeting of Conserved Gag-Epitopes in Early HIV Infection Is Associated with Lower Plasma Viral Load and Slower CD4+T Cell Depletion-Reference-Cited by-同舟云学术

Targeting of Conserved Gag-Epitopes in Early HIV Infection Is Associated with Lower Plasma Viral Load and Slower CD4+T Cell Depletion

Published:2013-03 Issue:3 Volume:29 Page:602-612
ISSN:0889-2229
Container-title:AIDS Research and Human Retroviruses
language:en
Short-container-title:AIDS Research and Human Retroviruses

Author:

Perez Carina L.¹²,Milush Jeffrey M.³,Buggert Marcus⁴,Eriksson Emily M.³,Larsen Mette V.⁵,Liegler Teri⁶,Hartogensis Wendy⁷,Bacchetti Peter⁸,Lund Ole⁵,Hecht Frederick M.⁷,Nixon Douglas F.³,Karlsson Annika C.⁴

Affiliation:

1. Department of Virology, Swedish Institute for Communicable Disease Control, Solna, Sweden.

2. Department of Microbiology, Tumor and Cell Biology, Karolinska Institutet, Stockholm, Sweden.

3. Division of Experimental Medicine, Department of Medicine, San Francisco General Hospital, University of California, San Francisco, California.

4. Department of Laboratory Medicine, Division of Clinical Microbiology, Karolinska Institutet, Stockholm, Sweden.

5. Department of Systems Biology, Center for Biological Sequence Analysis, Technical University of Denmark, Lyngby, Denmark.

6. Department of Medicine, University of California, San Francisco, California.

7. Positive Health Program, Department of Medicine, San Francisco General Hospital, University of California, San Francisco, California.

8. Department of Epidemiology and Biostatistics, University of California, San Francisco, California.

Publisher

Mary Ann Liebert Inc

Subject

Infectious Diseases,Virology,Immunology

Link

http://www.liebertpub.com/doi/pdf/10.1089/aid.2012.0171

Reference44 articles.

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4. Combination of Immune and Viral Factors Distinguishes Low-Risk versus High-Risk HIV-1 Disease Progression in HLA-B*5701 Subjects

5. Selective Escape from CD8 + T-Cell Responses Represents a Major Driving Force of Human Immunodeficiency Virus Type 1 (HIV-1) Sequence Diversity and Reveals Constraints on HIV-1 Evolution

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