Transgenic Expression of Antioxidant Protein Thioredoxin in PancreaticβCells Prevents Progression of Type 2 Diabetes Mellitus-Reference-Cited by-同舟云学术

Transgenic Expression of Antioxidant Protein Thioredoxin in PancreaticβCells Prevents Progression of Type 2 Diabetes Mellitus

Published:2008-01 Issue:1 Volume:10 Page:43-50
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Yamamoto Mayu¹,Yamato Eiji¹,Shu-Ichi Toyoda¹,Tashiro Fumi¹,Ikegami Hiroshi²,Yodoi Junji³,Miyazaki Jun-Ichi¹

Affiliation:

1. Division of Stem Cell Regulation Research, Osaka University Graduate School of Medicine, Osaka, Japan.

2. Department of Geriatric Medicine, Osaka University Graduate School of Medicine, Osaka, Japan.

3. Department of Biological Responses, Institute for Frontier Medical Sciences, Kyoto University, Kyoto, Japan.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2007.1586

Reference26 articles.

1. Oxidative Stress and Stress-Activated Signaling Pathways: A Unifying Hypothesis of Type 2 Diabetes

2. Are Oxidative Stress−Activated Signaling Pathways Mediators of Insulin Resistance and β-Cell Dysfunction?

3. Does the Glucose-Dependent Insulin Secretion Mechanism Itself Cause Oxidative Stress in Pancreatic β-Cells?

4. In vivo prevention of hyperglycemia also prevents glucotoxic effects on PDX-1 and insulin gene expression.

5. Oxidative Stress-mediated, Post-translational Loss of MafA Protein as a Contributing Mechanism to Loss of Insulin Gene Expression in Glucotoxic Beta Cells

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