Vascular Oxidant Stress and Inflammation in Hyperhomocysteinemia-Reference-Cited by-同舟云学术

Vascular Oxidant Stress and Inflammation in Hyperhomocysteinemia

Published:2007-11 Issue:11 Volume:9 Page:1941-1958
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Papatheodorou Louisa¹,Weiss Norbert¹

Affiliation:

1. Department of Vascular Medicine, Medical Policlinic–City Campus, University of Munich Medical Center, Munich, Germany.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2007.1750

Reference203 articles.

1. Homocysteine enhances superoxide anion release and NADPH oxidase assembly by human neutrophils. Effects on MAPK activation and neutrophil migration

2. Co-expression of ICAM-1, VCAM-1, ELAM-1 and Hsp60 in human arterial and venous endothelial cells in response to cytokines and oxidized low-density lipoproteins

3. Effect of thiol oxidation and thiol export from erythrocytes on determination of redox status of homocysteine and other thiols in plasma from healthy subjects and patients with cerebral infarction

4. Hyperhomocysteinemia Activates Nuclear Factor-κB in Endothelial Cells via Oxidative Stress

5. Folic acid inhibits homocysteine-induced superoxide anion production and nuclear factor kappa B activation in macrophagesThis paper is one of a selection of papers published in this Special Issue, entitled Young Investigator's Forum.

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