Some peculiarities of the molecular pathogenesis of gastroenteritis caused by non-typhoidal Salmonella

Author:

Boichenko M. N.1ORCID,Bosh’Ian R. E.1ORCID,Budanova E. V.1ORCID,Usatova G. N.1ORCID

Affiliation:

1. Federal State Autonomous Educational Institution of Higher Education I. M. Sechenov First Moscow State Medical University, Ministry of Healthcare of the Russian Federation (Sechenov University)

Abstract

The development of gastroenteritis caused by non-typhoidal Salmonella (NTS) is mediated by the action of effectors that belong to the Type three secretory systems (T3SS) of Salmonella enterica. These effectors are expressed by Salmonella pathogenicity islands 1 and 2 located in the genome. The effectors of T3SS-1 provide invasion of salmonella into host cell, take part in the development of inflammatory diarrhea some and may act as a predisposing factor in oncogenesis leading to the development of colorectal cancer (CRC). The T3SS-2 effectors provide intracellular survival of bacteria in the host cell and evading from the host adaptive immune response that may lead to the prolonged persistence.

Publisher

LLC Global Media Technology

Subject

Gastroenterology,Hepatology

Reference33 articles.

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